Robert Centor MD MACP 🇮🇱
Robert Centor MD MACP 🇮🇱

@medrants

5 Tweets 16 reads Mar 07, 2022
1/ #UncleBob recently presented a patient who had a hyperkalemia, normal gap acidosis (type 4 RTA) to @DxRxEdu & @rabihmgeha
But why does hyperkalemia cause a normal gap acidosis?
@tony_breu
2/ Some basic physiology - we metabolize around 1 mEq of H+ daily from our diet. We buffer that acid using titratable (phosphate) and non-titratable (NH4+) acids.
The phosphate pathway does not vary much, but our kidneys can normally control the ammonium pathway
3/ Where does the ammonia come from? Glutamine -> glutamate under the enzyme glutaminase produces NH3
Here is the interesting part. Increased K inhibits this enzyme, thus we produce insufficient NH3 to buffer our dietary intake.
4/ (interesting that low K stimulates the enzyme) which may contribute to hepatic encephalopathy - probably explains with low K is a risk factor for hepatic encephalopathy.
5/ Thus, if we can return the K to normal, the normal gap acidosis will resolve.
Here is a reference jasn.asnjournals.org
Mechanism of Hyperkalemia-Induced Metabolic Acidosis

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